Failure to clear cellular debris from the infarct compromises the endothelial lining of the left ventricle and exposes the damaged tissue to the blood.
The division begins in the common ventricle where a furrow in the outer surface of the heart will appear the interventricular foramen eventually disappears.
A heart that undergoes dilated cardiomyopathy shows unique enlargement of ventricles, and thinning of the ventricular wall that may lead to heart failure.